statistiche ricercatore

Continente	#
NA - Nord America	1.273
AS - Asia	681
EU - Europa	444
SA - Sud America	66
Continente sconosciuto - Info sul continente non disponibili	45
AF - Africa	13
Totale	2.522

Nazione	#
US - Stati Uniti d'America	1.236
SG - Singapore	247
CN - Cina	179
IT - Italia	104
DE - Germania	77
VN - Vietnam	75
GB - Regno Unito	65
BR - Brasile	53
PL - Polonia	51
HK - Hong Kong	44
FI - Finlandia	33
TR - Turchia	33
UA - Ucraina	31
CA - Canada	24
IN - India	24
FR - Francia	19
ID - Indonesia	18
SE - Svezia	18
JP - Giappone	16
BE - Belgio	12
RU - Federazione Russa	11
BD - Bangladesh	8
MX - Messico	8
AR - Argentina	5
MY - Malesia	5
NL - Olanda	5
EC - Ecuador	4
IQ - Iraq	4
PK - Pakistan	4
UZ - Uzbekistan	4
ZA - Sudafrica	4
AT - Austria	3
CR - Costa Rica	3
CZ - Repubblica Ceca	3
JO - Giordania	3
KE - Kenya	3
KR - Corea	3
SA - Arabia Saudita	3
AL - Albania	2
AZ - Azerbaigian	2
CO - Colombia	2
DZ - Algeria	2
ES - Italia	2
ET - Etiopia	2
KG - Kirghizistan	2
PH - Filippine	2
RO - Romania	2
AF - Afghanistan, Repubblica islamica di	1
BG - Bulgaria	1
CH - Svizzera	1
GF - Guiana Francese	1
HR - Croazia	1
IL - Israele	1
IR - Iran	1
JM - Giamaica	1
KZ - Kazakistan	1
LR - Liberia	1
LT - Lituania	1
NG - Nigeria	1
PE - Perù	1
PT - Portogallo	1
RS - Serbia	1
TH - Thailandia	1
TT - Trinidad e Tobago	1
XK - ???statistics.table.value.countryCode.XK???	1
Totale	2.478

Nome	#
An exon-specific U1snRNA induces a robust factor IX activity in mice expressing multiple human FIX splicing mutants	307
Regulation of a strong F9 cryptic 5'ss by intrinsic elements and by combination of tailored U1snRNAs with antisense oligonucleotides	303
Exploring Splicing-Switching Molecules For Seckel Syndrome Therapy	287
Genetic determinants of activated factor VII antithrombin complex plasma concentration include tissue factor, factor VII and endothelial protein C receptor gene variants	252
A unique exon specific U1snRNA rescues different haemophilia B - causing splicing-defective factor IX variants in mice	229
Correction of aberrant splicing causing haemophilia B through the combination of compensatory U1snRNAs and antisense oligonucleotides	211
A Compensatory U1snRNA Partially Rescues FAH Splicing and Protein Expression in a Splicing-Defective Mouse Model of Tyrosinemia Type I	190
Disease-causing variants of the conserved+2T of 5 ' splice sites can be rescued by engineered U1snRNAs	189
Rescue of missense and splicing mutations in Haemophilia A by a unique Exon Specific U1snRNA	188
The somatic FAH C.1061C>A change counteracts the frequent FAH c.1062+5G>A mutation and permits U1snRNA-based splicing correction	186
Regulation of exon definition by intrinsic elements and by combination of tailored U1snRNA with antisense oligonucleotides	180
Totale	2.522

	Totale	Lug	Ago	Sett	Ott	Nov	Dic	Gen	Feb	Mar	Apr	Mag	Giu
2021/2022	112	0	0	1	6	5	8	9	3	6	12	8	54
2022/2023	146	15	10	10	17	21	14	13	15	14	1	13	3
2023/2024	77	2	9	0	1	5	13	4	15	1	1	1	25
2024/2025	366	6	7	35	20	41	39	12	7	76	36	48	39
2025/2026	897	85	24	75	95	128	62	156	57	75	97	29	14
2026/2027	102	20	55	27	0	0	0	0	0	0	0	0	0
Totale													2.522

SFERA Archivio dei prodotti della Ricerca dell'Università di Ferrara