Toxoplasmosis is a widespread infection in nature that affects both humans and animals and is caused by the intracellular parasite, Toxoplasma gondii. The disease can be congenital or acquired. Ocular toxoplasmosis can represent a recurrence of congenital disease or can be secondary to postnatal infection. The hallmark of the disease includes focal necrotizing retinochoroiditis that ultimately results in a characteristic adjacent or nearby retinochoroidal atrophic scar. However, a variety of less common, "atypical" presentations may occur, eg, punctate outer retinal toxoplasmosis, retinal vasculitis, retinal vascular occlusions, rhegmatogenous and serous retinal detachments, unilateral pigmentary retinopathy mimicking retinitis pigmentosa, neuroretinitis and other forms of optic neuropathy, and scleritis. Although in the past most cases of ocular toxoplasmosis were considered to be the result of the reactivation of congenital infections, it has been ascertained that many postnatally acquired infections are also responsible for the ocular involvement.

BILATERAL NEURORETINITIS IN A 6-YEAR-OLD BOY WITH ACQUIRED TOXOPLASMOSIS.

SEBASTIANI, Adolfo;PARMEGGIANI, Francesco;
2003

Abstract

Toxoplasmosis is a widespread infection in nature that affects both humans and animals and is caused by the intracellular parasite, Toxoplasma gondii. The disease can be congenital or acquired. Ocular toxoplasmosis can represent a recurrence of congenital disease or can be secondary to postnatal infection. The hallmark of the disease includes focal necrotizing retinochoroiditis that ultimately results in a characteristic adjacent or nearby retinochoroidal atrophic scar. However, a variety of less common, "atypical" presentations may occur, eg, punctate outer retinal toxoplasmosis, retinal vasculitis, retinal vascular occlusions, rhegmatogenous and serous retinal detachments, unilateral pigmentary retinopathy mimicking retinitis pigmentosa, neuroretinitis and other forms of optic neuropathy, and scleritis. Although in the past most cases of ocular toxoplasmosis were considered to be the result of the reactivation of congenital infections, it has been ascertained that many postnatally acquired infections are also responsible for the ocular involvement.
Perrotta, S; Nobili, B; Grassia, C; Sebastiani, Adolfo; Parmeggiani, Francesco; Costagliola, C.
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Utilizza questo identificativo per citare o creare un link a questo documento: https://hdl.handle.net/11392/1207266
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